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normal chow diet (nd)  (Dyets Inc)

 
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    Dyets Inc normal chow diet (nd)
    Normal Chow Diet (Nd), supplied by Dyets Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/normal+chow+diet+(nd)/normal+chow+diet++ncd+/pm39647663-180-25-46
    Average 90 stars, based on 1 article reviews
    normal chow diet (nd) - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    Modification:

    Article Title: Amelioration of non-alcoholic fatty liver disease with NPC1L1-targeted IgY or n-3 polyunsaturated fatty acids in mice.
    Article Snippet: Patients with non-alcoholic fatty liver disease (NAFLD) have an increased risk for progression to hepatocellular carcinoma in addition to comorbidities such as cardiovascular and serious metabolic diseases; however, the current therapeutic options are limited.. Based on our previous report that omega-3 polyunsaturated fatty acids (n-3 PUFAs) can significantly ameliorate high fat diet (HFD)-induced NAFLD, we explored the therapeutic efficacy of n-3 PUFAs and N-IgY, which is a chicken egg yolk-derived IgY specific for the Niemann-Pick C1-Like 1 (NPC1L1) cholesterol transporter, on NAFLD in mice.. We generated N-IgY and confirmed its efficient cholesterol transport-blocking activity in HepG2 and Caco-2 cells, which was comparable to the effect of ezetimibe (EZM).

    Purification:

    Article Title: Amelioration of non-alcoholic fatty liver disease with NPC1L1-targeted IgY or n-3 polyunsaturated fatty acids in mice.
    Article Snippet: Patients with non-alcoholic fatty liver disease (NAFLD) have an increased risk for progression to hepatocellular carcinoma in addition to comorbidities such as cardiovascular and serious metabolic diseases; however, the current therapeutic options are limited.. Based on our previous report that omega-3 polyunsaturated fatty acids (n-3 PUFAs) can significantly ameliorate high fat diet (HFD)-induced NAFLD, we explored the therapeutic efficacy of n-3 PUFAs and N-IgY, which is a chicken egg yolk-derived IgY specific for the Niemann-Pick C1-Like 1 (NPC1L1) cholesterol transporter, on NAFLD in mice.. We generated N-IgY and confirmed its efficient cholesterol transport-blocking activity in HepG2 and Caco-2 cells, which was comparable to the effect of ezetimibe (EZM).

    Positive Control:

    Article Title: Amelioration of non-alcoholic fatty liver disease with NPC1L1-targeted IgY or n-3 polyunsaturated fatty acids in mice.
    Article Snippet: Patients with non-alcoholic fatty liver disease (NAFLD) have an increased risk for progression to hepatocellular carcinoma in addition to comorbidities such as cardiovascular and serious metabolic diseases; however, the current therapeutic options are limited.. Based on our previous report that omega-3 polyunsaturated fatty acids (n-3 PUFAs) can significantly ameliorate high fat diet (HFD)-induced NAFLD, we explored the therapeutic efficacy of n-3 PUFAs and N-IgY, which is a chicken egg yolk-derived IgY specific for the Niemann-Pick C1-Like 1 (NPC1L1) cholesterol transporter, on NAFLD in mice.. We generated N-IgY and confirmed its efficient cholesterol transport-blocking activity in HepG2 and Caco-2 cells, which was comparable to the effect of ezetimibe (EZM).

    Western Blot:

    Article Title: Augmenter of liver regeneration (ALR) crotonylation assists in mitochondria-ER contact to alleviate hepatic steatosis.
    Article Snippet: This is a PDF file of an article that has undergone enhancements after acceptance, such as the addition of a cover page and metadata, and formatting for readability, but it is not yet the definitive version of record.. This version will undergo additional copyediting, typesetting and review before it is published in its final form, but we are providing this version to give early visibility of the article.. Please note that, during the production process, errors may be discovered which could affect the content, and all legal disclaimers that apply to the journal pertain.



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    Seven-week-old male Ldl receptor -deficient ( Ldlr -/- ) mice were treated with tsRNA-Glu-CTC antisense oligonucleotides (ASO) or control ASO twice per week for 7 weeks. The mice were maintained on a <t>normal</t> <t>chow</t> <t>diet</t> (ND) or switched to a high-cholesterol diet (HCD) starting at 8-week-old age. a The schematic of tsRNA-Glu-CTC ASO treatment experiment. Created in BioRender. Zhou, C. ( https://BioRender.com/rj9jwzr ). b – g Body weight ( n = 8,10,10) ( b ), representative photos of collected serum ( c ), serum total cholesterol ( n = 8,10,10) (left panel) and triglyceride ( n = 5,10,10) (right panel) levels ( d ), cholesterol levels of lipoprotein fractions (VLDL-C, LDL-C, and HDL-C) ( n = 8,10,10) ( e ), representative Oil-Red-O (top) and hematoxylin and eosin (bottom) stained liver sections (scale bar =100 μm) ( f ), and hepatic cholesterol ( n = 6,9,6) (left panel) and triglyceride ( n = 6,8,6) (right panel) contents ( g ) of Ldlr -/- mice treated with vehicle control, control ASO, or tsRNA-Glu-CTC ASO (data are shown as mean ± SEM, one-way ANOVA, Bonferroni multiple-comparison test). h Representative images of Oil-red-O-stained sections and quantitative analysis of the atherosclerotic lesion area at the aortic root of Ldlr -/- mice ( n = 7,8,8; mean ± SEM, one-way ANOVA, Bonferroni multiple-comparison test). i Correlation between serum total cholesterol levels and atherosclerotic lesion sizes in Ldlr -/- mice ( n = 16; Correlation was analyzed by Pearson correlation). j Representative images of immunofluorescence staining of CD68 (green) and α-smooth muscle actin (αSMA) (red) at the aortic root of Ldlr -/- mice (scale bar=200 µm). Quantitative analysis of staining area is displayed as indicated ( n = 6,6,6; mean ± SEM, one-way ANOVA, Bonferroni multiple-comparison test). k The expression levels of hepatic lipogenic genes of HCD-fed Ldlr -/- mice treated with tsRNA-Glu-CTC ASO or control ASO were analyzed by quantitative real-time PCR ( n = 6, mean ± SEM, two-tailed Student’s t-test). n represents the number of biological replicates (mice). VLDL-C very low-density lipoprotein cholesterol; LDL-C low density lipoprotein cholesterol; HDL-C high density lipoprotein cholesterol.
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    Seven-week-old male Ldl receptor -deficient ( Ldlr -/- ) mice were treated with tsRNA-Glu-CTC antisense oligonucleotides (ASO) or control ASO twice per week for 7 weeks. The mice were maintained on a normal chow diet (ND) or switched to a high-cholesterol diet (HCD) starting at 8-week-old age. a The schematic of tsRNA-Glu-CTC ASO treatment experiment. Created in BioRender. Zhou, C. ( https://BioRender.com/rj9jwzr ). b – g Body weight ( n = 8,10,10) ( b ), representative photos of collected serum ( c ), serum total cholesterol ( n = 8,10,10) (left panel) and triglyceride ( n = 5,10,10) (right panel) levels ( d ), cholesterol levels of lipoprotein fractions (VLDL-C, LDL-C, and HDL-C) ( n = 8,10,10) ( e ), representative Oil-Red-O (top) and hematoxylin and eosin (bottom) stained liver sections (scale bar =100 μm) ( f ), and hepatic cholesterol ( n = 6,9,6) (left panel) and triglyceride ( n = 6,8,6) (right panel) contents ( g ) of Ldlr -/- mice treated with vehicle control, control ASO, or tsRNA-Glu-CTC ASO (data are shown as mean ± SEM, one-way ANOVA, Bonferroni multiple-comparison test). h Representative images of Oil-red-O-stained sections and quantitative analysis of the atherosclerotic lesion area at the aortic root of Ldlr -/- mice ( n = 7,8,8; mean ± SEM, one-way ANOVA, Bonferroni multiple-comparison test). i Correlation between serum total cholesterol levels and atherosclerotic lesion sizes in Ldlr -/- mice ( n = 16; Correlation was analyzed by Pearson correlation). j Representative images of immunofluorescence staining of CD68 (green) and α-smooth muscle actin (αSMA) (red) at the aortic root of Ldlr -/- mice (scale bar=200 µm). Quantitative analysis of staining area is displayed as indicated ( n = 6,6,6; mean ± SEM, one-way ANOVA, Bonferroni multiple-comparison test). k The expression levels of hepatic lipogenic genes of HCD-fed Ldlr -/- mice treated with tsRNA-Glu-CTC ASO or control ASO were analyzed by quantitative real-time PCR ( n = 6, mean ± SEM, two-tailed Student’s t-test). n represents the number of biological replicates (mice). VLDL-C very low-density lipoprotein cholesterol; LDL-C low density lipoprotein cholesterol; HDL-C high density lipoprotein cholesterol.

    Journal: Nature Communications

    Article Title: A cholesterol-responsive hepatic tRNA-derived small RNA regulates cholesterol homeostasis and atherosclerosis development

    doi: 10.1038/s41467-025-67387-z

    Figure Lengend Snippet: Seven-week-old male Ldl receptor -deficient ( Ldlr -/- ) mice were treated with tsRNA-Glu-CTC antisense oligonucleotides (ASO) or control ASO twice per week for 7 weeks. The mice were maintained on a normal chow diet (ND) or switched to a high-cholesterol diet (HCD) starting at 8-week-old age. a The schematic of tsRNA-Glu-CTC ASO treatment experiment. Created in BioRender. Zhou, C. ( https://BioRender.com/rj9jwzr ). b – g Body weight ( n = 8,10,10) ( b ), representative photos of collected serum ( c ), serum total cholesterol ( n = 8,10,10) (left panel) and triglyceride ( n = 5,10,10) (right panel) levels ( d ), cholesterol levels of lipoprotein fractions (VLDL-C, LDL-C, and HDL-C) ( n = 8,10,10) ( e ), representative Oil-Red-O (top) and hematoxylin and eosin (bottom) stained liver sections (scale bar =100 μm) ( f ), and hepatic cholesterol ( n = 6,9,6) (left panel) and triglyceride ( n = 6,8,6) (right panel) contents ( g ) of Ldlr -/- mice treated with vehicle control, control ASO, or tsRNA-Glu-CTC ASO (data are shown as mean ± SEM, one-way ANOVA, Bonferroni multiple-comparison test). h Representative images of Oil-red-O-stained sections and quantitative analysis of the atherosclerotic lesion area at the aortic root of Ldlr -/- mice ( n = 7,8,8; mean ± SEM, one-way ANOVA, Bonferroni multiple-comparison test). i Correlation between serum total cholesterol levels and atherosclerotic lesion sizes in Ldlr -/- mice ( n = 16; Correlation was analyzed by Pearson correlation). j Representative images of immunofluorescence staining of CD68 (green) and α-smooth muscle actin (αSMA) (red) at the aortic root of Ldlr -/- mice (scale bar=200 µm). Quantitative analysis of staining area is displayed as indicated ( n = 6,6,6; mean ± SEM, one-way ANOVA, Bonferroni multiple-comparison test). k The expression levels of hepatic lipogenic genes of HCD-fed Ldlr -/- mice treated with tsRNA-Glu-CTC ASO or control ASO were analyzed by quantitative real-time PCR ( n = 6, mean ± SEM, two-tailed Student’s t-test). n represents the number of biological replicates (mice). VLDL-C very low-density lipoprotein cholesterol; LDL-C low density lipoprotein cholesterol; HDL-C high density lipoprotein cholesterol.

    Article Snippet: The mice were either fed a normal chow diet (ND) (LabDiet, PicoLab Rodent Diet 5053) for 7 weeks or fed a ND for 1 week and then switched to a HCD for 6 weeks.

    Techniques: Control, Staining, Comparison, Immunofluorescence, Expressing, Real-time Polymerase Chain Reaction, Two Tailed Test